Primary objective of the study is to test the effectiveness of two interventions (by comparison with a control intervention and by mutual comparison) in preventing the negative effects of critical incidents and chronic job demands on the outcomes:…
ID
Source
Brief title
Condition
- Other condition
- Anxiety disorders and symptoms
Synonym
Health condition
burnout
Research involving
Sponsors and support
Intervention
Outcome measures
Primary outcome
Main study parameters: The main study parameters are the incidence of symptoms
of Post Traumatic Stress Disorder, Burnout and Absenteeism four weeks post
incident.
Secondary outcome
Secondary endpoint is the score on the Trauma Screening Questionnaire two weeks
post incident.
Background summary
Only recently researchers have started to examine the impact of acute stressors
(critical incidents) in the workplace, next to chronic job stressors. High
rates of (symptoms of) Post Traumatic Stress Disorder (PTSD) have been reported
among health care workers in acute settings, which negatively impact their well
being and their attitude towards the job. Chronic job demands (e.g. demanding
contacts with patients, time constraints, poor communication) are positively
related to burnout (exhaustion and disengegement). With regard to the expected
shortage of health care workers in the Netherlands in the near the future,
impaired well being and disengagement may lead to serious problems; not only
for the individual health care worker, but also for the organization, and for
society. Preventive programs should protect and support health care
professionals after work-related critical incidents (Arbo-wet, 1994). Until now
however, no consensus exists with regard to the content of preventive
intervention after critical incidents.
Job resources (e.g. autonomy, social support, relationship with supervisor) are
considered health-protecting factors, that buffer the impact of high job
demands on burnout. Lack of resources leads to disengagement and indirectly to
short-term absenteeism. The impact of critical incidents on outcomes, like
posttraumatic responses, exhaustion, and absenteeism may also be buffered by
job resources (i.e. psycho education, social support, autonomy, support from
co-workers, and supervisor support). Besides, job resources may exert their
effect (partly) via personal resources (e.g. self-efficacy, self esteem,
resilience, optimism; see work model, page 21).
Neuro-endocrine response: In this study, the neuro-endocrine response to stress
(i.e. cortisol, and dehydroepiandrosterone/DHEA) is included as the more
objective measure of stress (next to self report questionnaires). Although, in
PTSD, hyper-regulated stress systems (low cortisol baseline level in
combination with over reactiveness to stress), as well as the opposite have
been found, DHEA to cortisol ratio may index the degree to which an individual
is buffered against the negative effects of stress.
Also in burnout-patients mixed results with respect to cortisol levels have
been found. However, when hormone levels and exhaustion symptoms were
simultaneously assessed (in a diary study), more severe burnout symptoms were
consistently associated with lower levels of cortisol, smaller Cortisol
Awakening Rise, and smaller cortisol to DHEA ratios.
Study objective
Primary objective of the study is to test the effectiveness of two
interventions (by comparison with a control intervention and by mutual
comparison) in preventing the negative effects of critical incidents and
chronic job demands on the outcomes: posttraumatic responses, exhaustion,
(dis)engagement, absenteeism, and satisfaction with help after critical
incidents.
Secondary objectives are:
a) to examine inter-correlations of critical incidents, chronic job demands,
job resources and personal resources, in inducing ASD/PTSD symptoms, general
health problems, depression, anxiety, substance abuse (tobacco, alcohol,
medication), exhaustion, disengagement, and absenteeism; across time, for the
three conditions (see model 1, p. 21).
b) to determine stress hormone levels in saliva, to explore associations
between DHEA to cortisol ratio in saliva and ASD/PTSD, burnout symptoms,
cognitive appraisal and coping (by simultaneous registration in a diary, thus
capturing states instead of constructs).
Study design
The study is longitudinal, randomized, *placebo* controlled (for flow chart,
see p. 25-26).
Intervention
The Controlgroup receives a *placebo* intervention (watchful waiting).
Interventiongroup-1 receives watchful waiting + psycho-education.
Interventiongroup-2 receives watchful waiting + psycho-education + incident
focussed social support from trained colleagues (see flow chart p 26, and
teratment of subjects section, page 26-37).
Study burden and risks
The expected burden mainly originates from completing the questionnaires
(appendices 1, 4, 5, 6), keeping a diary, and saliva sampling (appendix 9a -
9f). The risks associated with participation consist of extra workload for the
colleague-supporters. There may be a risk of initially having more instead of
fewer complaints, induced by higher awareness (as a result of psycho-education
and questionnaire-items). Although in the intervention groups, this is not what
we expect, as we explicitly label possible responses as normal reactions to
abnormal situations that will probably disappear in a few days to weeks.
dr. Molewaterplein 60
3000 CB Rotterdam
NL
dr. Molewaterplein 60
3000 CB Rotterdam
NL
Listed location countries
Age
Inclusion criteria
Meeting with a work-related critical incident (critical incidents will be further defined after a first inventarisation in the in D3 mentioned wards)
Working in the present ward for at least 6 months at T0
Exclusion criteria
Current serious psychiatric disorder, that needs treatment before any other intervention could be applied
Currently under treatment for any psychiatric problem
Design
Recruitment
Followed up by the following (possibly more current) registration
No registrations found.
Other (possibly less up-to-date) registrations in this register
No registrations found.
In other registers
Register | ID |
---|---|
CCMO | NL23132.078.08 |